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| young fat girls , hepatitis medication, plump fiction , amiodarone, inflammation, abdominal pain, cholesterol, aids, plump mature women , transfusion, blood, transfused, | The SREBPs are a family of transcription factors that are key regulators for cholesterol and fatty acid synthesis; they exert their effect by directly activating the expression of more plump than 30 plump genes in the liver. Recently, researchers have demonstrated that plump chronic ethanol administration can significantly increase the production of hepatic SREBP-1, which is associated with increased expression of lipogenic genes as well as accumulation of triglycerides in the liver. Using 4-methylpyrazole and cyanamide, it was shown that this effect of alcohol was dependent on its metabolism to acetaldehyde. |
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| Ethanol may inhibit CPT-1 activity by triggering a cascade of events starting from an activation of sterol regulatory element-binding hepatitis medication protein (SREBP) to up-regulation of acetyl Co-A carboxylase (ACC), and subsequent increased production of malonyl Co-A that is known to inhibit CPT-1 activity. Alternatively, ethanol may inhibit CPT-1 activity by triggering another cascade of events starting from inhibition of PPAR-á activity to inhibition of malonyl Co-A hepatitis medication decarboxylase (MCD) activity, and subsequent increased production of malonyl Co-A. d. Accelerated de novo Fatty Acid Synthesis: Chronic ethanol use and hepatitis medication metabolic stresses such as obesity and insulin resistance have been shown to stimulate lipogenesis in the liver through increased transcription of genes for lipogenic enzymes. |
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