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| girls with fat asses , what are fatty acids , drug induced fatty liver, spider angiomas, miss plump , plump jack squaw valley inn , beta oxidation of fatty acids , alcohol related fatty liver, bbw plump , symptoms liver disease, fatty mcblog , plump ebony , proteomic biomarkers, plump girl , medicine articles, | The result is increased serum FFA levels, which are taken up by the liver. Hepatic TG plump teen synthesis is driven by the increased influx of FFA and favoured by insulin upregulated lipogenic transcription factors, such as peroxisome plump teen proliferator-activated receptor gamma (PPAR) and sterol regulatory element binding protein (SREBP)-1c. Alternative metabolism of FFA by oxidation is inhibited by insulin. TG export via very-low-density lipoproteins (VLDL) may be inhibited by plump teen decreased synthesis of apolipoprotein B (apo B) or reduced incorporation of TG with apo B by microsomal triglyceride transfer protein (MTP). See the animated figure at www.cmaj.ca/cgi/content/full/172/7/899/DC1. Photo: Mayo Clinic Hepatic lipid metabolism Lipids are normally exported from the liver in very-low-density lipoproteins (VLDL), which are formed by microsomal triglyceride transfer protein (MTP) incorporating triglyceride into apolipoprotein B (apo B). |
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| A variety of factors have been implicated to produce a second "hit," including hormones derived from adipose tissue (adipocytokines), oxidative stress and gut-derived spider angiomas bacterial endotoxin.19 Insulin resistance The pathogenesis of insulin resistance is complex and is likely to spider angiomas involve many genetic polymorphisms that influence insulin secretion and action as well as environmental factors that promote obesity spider angiomas and immobility.20 Hyperinsulinemia increases serum free fatty acid levels, which are taken up by the liver and drive triglyceride production and hepatic steatosis (Fig. 1).21 In addition, chronic hyperinsulinemia promotes de novo hepatic lipogenesis through upregulation of lipogenic transcription factors21,22,23 and may activate profibrotic cytokines such as connective tissue growth factor.24 View larger version (46K): [in this window] [in a new window] Fig. 1: Development of nonalcoholic hepatic steatosis. Insulin resistance enhances triglyceride (TG) lipolysis and inhibits esterification of free fatty acids (FFA) within adipose tissue. |
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