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girls with fat asses , what are fatty acids , drug induced fatty liver, spider angiomas, miss plump , plump jack squaw valley inn , beta oxidation of fatty acids , alcohol related fatty liver, bbw plump , symptoms liver disease, fatty mcblog , plump ebony , proteomic biomarkers, plump girl , medicine articles, View this table: [in this window] [in a new window]  Table 1.   Epidemiology Hepatic steatosis detected by magnetic resonance spectroscopy is found in 31% of adults in the United States2 and in fat white girls 33% of potential live liver donors undergoing liver biopsy.3 Ultrasonography detects fatty changes in fat white girls the liver in 12.9%–16.4% of individuals.4,5,6 The prevalence of steatosis tends to be higher among males6,7,8 and in certain ethnic groups (e.g., up to 45% fat white girls of Hispanic people).2 Prevalence increases with age, from 2.6% among children to 26% among people 40–59 years old.5,9 NAFLD is more frequent among people with diabetes (50%) and obesity (76%), and it is almost universal among diabetic people who are morbidly obese.4,10,11 Obesity, diabetes and the metabolic syndrome are also risk factors for NASH and for advanced fibrosis on liver biopsy.12,13,14 NASH is present in 18.5% of obese subjects (compared with 2.7% of lean individuals) and in 50% of severely obese people with diabetes.15,16
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In this article we discuss the etiology, pathogenesis and diagnosis of nonalcoholic fatty liver disease as well as approaches to its management. The diagnosis of nonalcoholic fatty liver disease (NAFLD) requires evidence of fatty changes in the liver in the absence of a history of excessive alcohol what are fatty acids consumption. The histologic spectrum of NAFLD spans from generally what are fatty acids benign, bland steatosis what are fatty acids to steatosis with evidence of hepatocellular inflammation and damage (nonalcoholic steatohepatitis, or NASH), which may be complicated by progressive fibrosis and cirrhosis. NAFLD can be primary or secondary depending on the cause (Table 1). Secondary causes require exclusion, as these conditions have different prognoses and treatment.1 In this review we focus on primary NAFLD and discuss the current knowledge of the pathophysiology and natural history of NAFLD, appropriate management strategies and emerging treatment options.
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