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what is nonalcoholic fatty liver diseasee, seronegative, saturated and unsaturated fatty acids , alcohol/addiction/illegaldrugs news, weight loss, menstrual disorders, symptoms of nonalcoholic fatty liver diseasee, plump jack squaw valley , symptoms of fatty liver , hepatitis c virus, cocaine, liver disease treatment, hepatitis c treatment, fatty woman , varices, fatty girl lyrics , fattyliver disease becoming popular, hepatitis c, galactosemia, plump rumps , plump rumps tgp , Mortality is dependent upon histological stage. Biopsy Undistinguishable from alcoholic hepatitis Significant lorenzo bertolini fibrosis in 39 lorenzo bertolini % (1) Cirrhosis in 8 - 17 % lorenzo bertolini (1,13,14,18, 31) Iron overload frequently seen (8) Predictors of fibrosis: Age, obesity, diabetes and AST/ALT > 1 (31) Treatment Recent studies identify a potential benefit of PPAR-g ligands on liver enzymes and histology (); the price in all studies was weight gain. Betaine, ursodeoxycholic acid, benzofibrates and N-acetyl cysteine could be beneficial but need further study in controlled trials (38).  Weight loss (6), eventually surgically induced (22) Phlebotomies in iron overloaded patients (37) Liver transplantation for cirrhosis; cave recurrence See current trials at the department Features of NASH in a recent, large series (35) TG and aHTN stand for hypertriglyceridemia and arterial hypertension, respectively Further links with respect to NASH Novel aspects of NASH Artikel zur Fettleber (pdf; in German) Dr.
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Female sex, morbid overweight and diabetes are accepted risk factors for progression (1,19). Recently, high angiotensin and TGF-b producing polymorphisms together have been described to predict fibrosis with an OR of 5.7 (42). There is reversal after weight loss (6,22). On the other hand, several cases of recurrence after liver transplantation have been described (2,11,15). cocaine Histological progression has been observed in 38 % of patients after a mean follow-up of 3.5 years (13); among these 49 patients, however, only 1 showed hepatic decompensation and 15 % progressed to cocaine cirrhosis. In contrast, Powell and colleagues observed progression in only 5 % of their patients (19) while Teli et al. could not observe progression at all (24). Cryptogenic cirrhosis is probably the end-result of NASH; such patients have the same incidence of decompensation and hepatocellular carcinoma than patients with hepatitis C-related or lean cryptogenic cirrhosis (40).
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