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These data support a role for adiponectin in protection hepatitis c medication against liver injury, in keeping with the hypothesis that an imbalance between proinflammatory and anti-inflammatory cytokines may have a pathogenic role in the development of liver damage in NAFLD. Finally, a novel cross-talk between adipose tissue and liver is emerging that may act as a major player in the link between metabolic syndrome and fatty liver disease. Acknowledgements This work was supported by a grant from the University of Padova (ex-60% hepatitis c medication 2002) to C P, and by a hepatitis c medication grant from the Italian MIUR (Ministero dell’Istruzione, dell’Universita ‘ e della Ricerca, COFIN n. 2003061834-006) to R V. The expert technical assistance of Mrs Marilena Tormene and Mrs Sonia Leandri is gratefully acknowledged. References Top Abstract Introduction Patients and methods Results Discussion References Received 17 August 2004 Accepted 5 October 2004 This article has been cited by other articles: (Search Google Scholar for Other Citing Articles) C.
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