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| what is nonalcoholic fatty liver diseasee, seronegative, saturated and unsaturated fatty acids , alcohol/addiction/illegaldrugs news, weight loss, menstrual disorders, symptoms of nonalcoholic fatty liver diseasee, plump jack squaw valley , symptoms of fatty liver , hepatitis c virus, cocaine, liver disease treatment, hepatitis c treatment, fatty woman , varices, fatty girl lyrics , fattyliver disease becoming popular, hepatitis c, galactosemia, plump rumps , plump rumps tgp , | Etiology Diabetes mellitus, in particular insulin resistance syndrome; insulin resistance is even found in lean subjects with normal glucose tolerance (30). Morbid overweight Hyperlipoproteinemia Drugs including tamoxifen proteomic technologies (27) Long-term steroid (10) or "massive" estrogen (20) treatment Bacterial overgrowth (16) Jejuno-ileal bypass (3,17) No identifiable proteomic technologies factor(s) in 42 % of patients (1) a1-antitrypsin deficiency MS (29) HFE proteomic technologies mutations? (28) Common genetic risk since it occurs in families (35); some affected family members are found to have 'cryptogenic' cirrhosis (32). Natural history Poorly understood. |
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| In favor hepatitis c virus of this finding may be the fact that cytochrome P450 2E1 is induced as in alcoholic liver disease (25). Decreased activity of short- and long- chain 3-hydroxyacyl-CoA dehydrogenase in liver biopsies (5,12) while b-oxidation is normal (5). All parts of the respiratory chain were decreased by about 30 %, the decrease correlating with BMI and insulin resistance (41). Heterozygosity for C282Y has been implicated (8). Indeed, different mutations in the HFE gene occur more frequently in NASH patients than in controls in patients of Angloceltic origin hepatitis c virus (28, 39). In obese animals there is increased intestinal production of ethanol (33). The pathogenetic role of this in man remains to be explored. Intestinal bacterial overgrowth resulting in increased TNF-a levels (34). Recently, hyperglycemia and -insulinemia has been implicated to be directly fibrogenetic via stimulation of connective tissue growth factor (36). |
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