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| steatosis, sirrohsis, roferon a, pictures of fat girls , health news, pegasys combination therapy, fibrotest, hepatomegaly, jaundice, plump and busty 2 , proteomics services, stefano rodella, 24 7 fat girls , fatty acid analysis , sexy plump , genotype, | b. Impaired Fatty Acid Oxidation: Alcohol and excess nfld fatty acids have been shown to inhibit fatty acid oxidation in liver slices from rats and humans, in perfused rat liver, in nfld rat hepatocytes, and in vivo in humans. Various mechanisms have been proposed for this effect. A redox shift resulting in increased ratio of NADH/NAD+ has been implicated in the development of fatty liver via inhibition nfld of mitochondrial fatty acid beta oxidation and tricarboxylic acid cycle. However, normalization of redox state failed to attenuate alcohol-induced fatty liver, suggesting that other mechanisms may also be contributing to this condition. Alcohol and excess fatty acids may also impair fatty acid oxidation by inhibiting the activities of enzymes involved in fatty acid oxidation. |
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| Proposals investigating the modulating effects of dietary fatty acids, obesity, diabetes, and insulin resistance on the development of alcoholic and nonalcoholic fatty liver are also encouraged. a. Increased Hepatic Uptake of genotype Fatty Acids : Fatty acid levels are significantly increased in the liver after alcohol consumption and in persons with obesity and the metabolic syndrome. Alcohol may increase hepatic fatty acid uptake by increasing hepatic blood flow. In addition, studies with isolated hepatocytes and perfused livers have suggested that alcohol can directly genotype increase fatty acid uptake independent of blood flow. This could be due to a direct effect of alcohol on the physical properties of the hepatic plasma membrane consequent to altered lipid composition. Further studies are required to delineate the role of fatty acids and the modulatory effect of alcohol and the metabolic syndrome upon hepatic uptake of fatty acids. |
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