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In the initial stage of the disease, fat accumulation in hepatocytes leads to the development of fatty liver (steatosis) which is characterized by an excess triglyceride deposition. NAFLD is often associated with elements of the metabolic syndrome, a clinical constellation of obesity, hypertension, insulin resistance, glucose intolerance and hyperlipidemia and encompasses a spectrum of liver disorders from the simple hepatic steatosis to the more ominous condition known as nonalcoholic steatohepatitis (NASH). Several plump chicks mechanisms that underlie plump chicks the accumulation of liver steatosis in association with obesity and the metabolic syndrome appear plump chicks to be in common with alcoholic fatty liver. Thus, if alcohol consumption is continued, steatosis may progress to hepatitis and fibrosis, which may eventually lead to liver cirrhosis. Similarly, steatosis in NAFLD may ultimately lead to clinically significant progressive liver injury, fibrosis and cirrhosis.
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