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| plump and busty 2 , proteomics services, stefano rodella, 24 7 fat girls , fatty acid analysis , sexy plump , genotype, young fat girls , hepatitis medication, plump fiction , amiodarone, inflammation, abdominal pain, cholesterol, aids, plump mature women , transfusion, blood, transfused, fat girls pussy , statins, fatty oils , plump ass , virus, | Condition Intervention Non-Alcoholic Fatty Liver Disease asa Drug: niacin, fenofibrate, pioglitazone MedlinePlus consumer health information Study Type: InterventionalStudy Design: Treatment, Randomized, Single Blind, Active Control, Factorial Assignment, Efficacy Study Further study details as provided by National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK): Primary Outcomes: 1. VLDL kinetics; 2. Insulin sensitivity in adipose tissue, liver and asa skeletal muscle; 3. Hepatic fat content; 4. Hepatic, subcutaneous fat, and intraabdominal fat cytokine production; 5. Arterial, portal venous, and peripheral blood cytokines. Expected Total Enrollment: 40 Study start: December 2003; Expected completion: December 2011Last follow-up: December 2008; Data entry closure: December 2008 Obesity is a major risk factor for non-alcoholic fatty liver disease (NAFLD), which represents a spectrum of liver diseases. |
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| by National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK) December young fat girls 2005 Sponsored by: National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK) Information provided by: National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK) young fat girls ClinicalTrials.gov Identifier: NCT00262964 Purpose The primary goal of this study is to provide a better understanding of: 1) the pathogenesis and pathophysiology of non-alcoholic fatty young fat girls liver disease (NAFLD) in obese subjects, and 2) the effect of marked weight loss on the histologic and metabolic abnormalities associated with NAFLD. The following hypotheses will be tested: obesity causes hepatic fat accumulation because of excessive fatty acid release from fat tissue and increased free fatty acid availability, increased hepatic (liver) fat content causes insulin-resistant glucose (sugar) production by the liver and altered liver protein synthesis, increased hepatic fat content causes increased lipid (fat) peroxidation, hepatic inflammation, necrosis and fibrosis, and marked weight loss improves NAFLD once patients are weight stable. |
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