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| auto immune, oxidative stress, non alcoholicsteatohepatitis, plump and busty , proteomic diagnostics, hepatocellular carcinoma, lose weight, fat bottom girls lyrics , plump butt , health, celiacdisease, lipids, fatty tumors in dogs , diabetes, omega minus three fatty acids , hepatic steatosis, | By using the glucose clamp technique, we intended to estimate insulin sensitivity quantitatively and validate previous results obtained by HOMA calculation (17). The HOMA measure, which considers only glucose and insulin concentrations under fasting, nondynamic conditions, is expected protein microarrays to give a picture of basal insulin effects, mainly on the liver, considering the importance of protein microarrays HGP on fasting hyperglycemia (35). The euglycemic clamp, measuring glucose disposal under hyperinsulinemia and unlimited substrate availability, gives a broader evaluation of insulin sensitivity and, when coupled with tracer methodology, more clearly defines the site of insulin resistance. protein microarrays We showed that in NAFLD patients, HGP is normal or high-normal in the fasting state, despite basal hyperinsulinemia, and that the liver does not switch off glucose production in response to insulin infusion. |
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| Our data plump and busty are supported by the recent finding that fasting hyperinsulinemia and reduced glucose tolerance, markers of plump and busty insulin resistance, are present in both obese and normal-weight NAFLD patients (30), although the total amount of hepatic fat deposition seems to be proportional to the degree of obesity (6). NAFLD, irrespective of obesity and fasting glucose levels, might be included in the metabolic syndrome (31), having obesity and type 2 diabetes as main clinical features (14), but also associated with raised triglyceride levels, hypertension (32), and splanchnic fat distribution (33). Previous studies have associated liver disease and raised transaminases to plump and busty the metabolic syndrome only in severe obesity (34), suggesting that NAFLD might be a secondary phenomenon. In the present study, the association of fatty liver with features of the metabolic syndrome was independent of BMI. Most patients, including lean patients, had a family history of diabetes or hypertension, fitting the proposed criteria for the insulin-resistance syndrome (31) and pointing to a genetic defect, sometimes magnified by other phenotypic expressions. |
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