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was nearly halved liverdisease/hepatitis news in a series of biopsy-proven NAFLD patients. To rule out diabetes as a primary cause of insulin resistance, only NAFLD subjects with normal fasting glucose and glucose tolerance were selected for the present study. We also ruled out the liverdisease/hepatitis news possibility that obesity played a role; although total fat mass and intra-abdominal fat were not quantitatively measured, 33% of patients had a normal body weight. Recalculating glucose disposal per kilogram of lean body mass (LBM), predicted by liverdisease/hepatitis news the dated Hume formula (29), does not quantitatively change the results (50.4 ± 9.1 vs. 27.5 ± 4.8 vs. 27.0 ± 8.8 µmol · kg-1 LBM · min-1 in control subjects, diabetic patients, and NAFLD patients, respectively, by ANOVA; P < 0.0001). This quantitative analysis was confirmed by the lack of difference in glucose disposal between normal- and overweight NAFLD subjects and the lack of correlation between BMI and glucose disposal in the NAFLD series.
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