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| ldl c, american surgical association, starvation, fatty fucking , fat girls and feeders , queen fat bottom girls , pleasingly plump , hcv treatments, impaired liver function, plump fuck , plump girls galleries , immunization, refsum disease, poynard, plump buttocks , liverdisease, plump djs , iv drugs, plump teens , steatosis, | In order to understand hepatic fat accumulation, a brief review of hepatic lipid metabolism under normal conditions is presented. Under normal hey fatty boom boom fed conditions, dietary triglycerides and surplus carbohydrates are converted into free fatty acids. (FFA). These fatty acids are transported to the liver hey fatty boom boom and stored as re-esterified triglycerides in adipose tissue nr used as energy sources by muscle. During periods of fasting or starvation, the triglycerides stored in adipose tissue are hydrolyzed to FFAs and transported to the liver, where they are used to form phospholipids and cholesterol esters or hey fatty boom boom converted into ketone bodies to be used as fuel by extrahepatic tissues.9 |
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| NASH is defined as the combination of steatosis with lobular inflammation and hepatocyte ballooning with/without hepatocyte necrosis. Most patients with NASH will have some iv drugs degree of fibrosis and some 20% will have cirrhosis. Angulo et al. reported that in patients with NASH, 74% will have iv drugs fibrosis and 20% will have cirrhosis at the time of their initial liver biopsy.4 The clinical and serologic findings of patients with NAFLD do not correlate well with the histologic findings. Currently, the best known predictors of advanced fibrosis are obesity, type 2 diabetes mellitus, and iv drugs an age greater than 45. Pathogenesis The pathogenesis of NASH is not well understood. Currently, the leading hypothesis in the pathogenesis of NASH is a two-hit theory consisting of hepatic fat accumulation owing to insulin resistance followed by oxidative stress arising from ROS produced primarily by mitochondria.8 |
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