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hyperlipidemia, cme, polyethylene glycol, omega 3 fatty acid fish oil , peginterferon alpha 2a, ribavirin interferon, nonalcoholic steatohepatitis, side effects, lemagazine, plump booty , sirrosis, seropositive,
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This can occur when the amount of fatty acid supplied to aspirin the liver from the gut or adipose tissue exceeds the amount needed aspirin for mitochondrial oxidation, phospolipid and cholesterol ester synthesis[5,11]. This is the presumed mechanism for aspirin steatosis in the setting of diabetes mellitus, obesity, malnutrition, acute starvation, total parenteral nutrition, steroid treatment and excessive dietary intake of fats[3,5,24]. Hyperinsulinemia and insulin resistance may be important components in the development of steatosis in these diseases[6,25]. In hyperinsulinemia, fatty acids are esterified to triglycerides[25]. In the study performed by Rashid et al.[6], acanthosis nigricans, which is a cutaneous marker of hyperinsulinemia, was detected in 13 of 36 patients with non-alcoholic fatty liver. Similarly, acanthosis nigricans was detected in 10 patients with fatty liver in our study. The relation between diabetes and steatohepatitis is clearly known[25]. Therefore, we did not include diabetic patients in our study.
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