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| hcv medication, liver, plump dj , vitamin e, treatment, seroprevalence, hcv virus, cirrhosis, ldl cholesterol, infection, combination therapy, information, big fat girls , ncbi, plump video , bobby plump , nonalcoholic fatty liver diseasee treatments, sexy plump women , omega3 fatty acids , plump gallery , plump asian , chronic liver inflammation, peglation, cholangiocarcinoma, | As regards the cause of extracellular matrix deposition in liver tissue, the hepatic sinusoidal lumens are narrowed by swollen hepatocytes containing plump breasts fat droplets and abundant enlarged endoplasmic reticula in their cytoplasm, and the hepatic sinusoidal microcirculation is impaired. Consequently, ischemia occurs in the perivenular or intralobular spaces, and hepatic fibrogenesis plump breasts appears to be enhanced by ischemia, which plump breasts may lead to liver cirrhosis16. Conclusion Diet therapy can decrease body weight, Body Mass Index (BMI) and liver biochemical abnormalities and therefore may help to improve Non-Alcoholic Fatty Liver Disease (NAFLD). Acknowledgment:This study was supported by grant No.1718 from Research Council of Shiraz University of Medical Sciences. The authors wish to thank vice-chancellor for research affairs. |
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| The primary metabolic abnormalities leading to lipid accumulation are not well understood, but they could consist seroprevalence of alterations in the pathways of uptake, synthesis, degradation, or secretion in hepatic lipid metabolism resulting from insulin resistance. Insulin resistance owing to inhibition of tumor necrosis factor-a (TNF-a) leads to the accumulation of fat in hepatocytes by two main mechanisms: Lipolysis, which increases seroprevalence circulting fatty acids and hyper insulinemia. Increased uptake of fatty acids by hepatocytes seroprevalence leads to mitochondrial -oxidation overload, with the consequent accumulation of fatty acids within hepatocytes. Hyperinsulinemia resulting from insulin resistance increases the synthesis of fatty acids in hepatocytes by increasing glycolysis and favors the accumulation of triglycerides within hepatocytes by decreasing hepatic production of apolipoprotein B-10017. Since weight reduction leads to decrease in TNF-a and other factors it may improve the liver disease. |
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