CP, da Costa Gayotto fibrosis celiacdisease

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oxidative stress, non alcoholicsteatohepatitis, plump and busty , proteomic diagnostics, hepatocellular carcinoma, lose weight, fat bottom girls lyrics , plump butt , health, celiacdisease, lipids, fatty tumors in dogs , diabetes, Samples of plasma and liver were submitted to biochemical, histological and oxidative stress analysis. Variables measured included serum levels of aminotransferases (AST, ALT), cholesterol and triglycerides. Oxidative stress was measured by lucigenin-enhanced luminescence and the concentration of hydroperoxides (CE-OOH-cholesteryl ester) in the liver tissue. RESULTS: We observed moderate macro- and fibrosis microvesicular fatty change in periportal fibrosis zones G1 and G2 as compared to controls (G3). In G2, fatty change was more severe. The inflammatory infiltrate was fibrosis scanty and no fibrosis was seen in any group. There was a significant increase of AST and triglycerides in G1 and G2 as compared to control group G3. The lucigenin-amplified luminescence (cpm/mg/min x 10(3)) was significantly increased in G1 (1393-/+790) and G2 (7191-/+500) as compared to controls (513-/+170), p<0.05. The concentrations of CE-OOH were higher in G1 (5.7-/+0.9 nmol/mg protein) as compared to control (2.6-/+0.7
CP, da Costa Gayotto LC, Tatai C, Della Bina BI, Janiszewski M, Lima ES, Abdalla DS, Lopasso FP, Laurindo FR, Laudanna AA.Deptartment of Gastroenterology of the Medical School, University of Sao Paulo, Instituto Central, Sao Paulo, celiacdisease Brazil. mauclaud@uol.com.brBACKGROUND/AIM: The pathogenesis of Nonalcoholic Fatty Liver Disease remains celiacdisease largely unknown, but oxidative stress seems to be involved. The aim of this study was to evaluate the role of oxidative stress in experimental celiacdisease hepatic steatosis induced by a choline-deficient diet. METHODS: Fatty liver disease was induced in Wistar rats by a choline-deficient diet. The animals were randomized into three groups: I (G1) and II (G2), n=6 each--fed with a choline-deficient diet for four and twelve weeks respectively; Group III (control-G3; n=6)--fed with a standard diet for twelve weeks.
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